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The longevity paradox: How to die young at a ripe old age

Analysis by Dr. Joseph Mercola Fact Checked

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  • Certain plant lectins are highly inflammatory and interfere with nutrient absorption across your intestinal wall. Evidence suggests avoiding these lectins could help boost longevity by protecting gut health
  • Many of those 95 or older who are still in excellent health have an IGF-1 level between 70 and 80. Restricting animal protein has been shown to consistently lower IGF-1 levels to a significant degree
  • IGF-1 activates the mTOR pathway, which inhibits autophagy, the breakdown and repair of damaged subcellular parts. Keeping IGF-1 low will allow you to properly cycle through autophagy activation
  • Metabolic inflexibility also inhibits autophagy, which is a necessary process for optimal health and longevity. A ketogenic diet and cyclical fasting are both effective ways to regain the metabolic flexibility to burn both sugar and fat for fuel
  • Polyphenols such as quercetin, fisetin, EGCG or resveratrol can be used to activate and augment autophagy

In this interview, Dr. Steven Gundry, medical director for International Heart & Lung Institute and director for Center for Restorative Medicine, reviews some of the concepts covered in his book “The Longevity Paradox: How to Die Young at a Ripe Old Age.”

While trained as a cardiothoracic surgeon, Gundry now specializes in treating patients holistically, focusing on food. I’ve previously interviewed him about plant lectins — the topic of his book “The Plant Paradox” — and the damage they can wreak on your health. Considering their potential influence, it’s not surprising to find plant lectins covered in his book on longevity as well.


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